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+Tissue inhibitor of metalloproteinase-3 is up-regulated by transforming growth factor-beta1 in vitro and expressed in fibroblastic foci in vivo in idiopathic pulmonary fibrosis
+Upregulation of alveolar levels of activin B, but not activin A, in lungs of west highland white terriers with idiopathic pulmonary fibrosis and diffuse alveolar damage
+Progressive transforming growth factor beta1-induced lung fibrosis is blocked by an orally active ALK5 kinase inhibitor
+Upregulation of activin-B and follistatin in pulmonary fibrosis - a translational study using human biopsies and a specific inhibitor in mouse fibrosis models
+Resveratrol inhibits transforming growth factor-b-induced proliferation and differentiation of ex vivo human lung fibroblasts into myofibroblasts through ERK/Akt inhibition and PTEN restoration
+Interactions between b-catenin and transforming growth factor-b signaling pathways mediate epithelial-mesenchymal transition and are dependent on the transcriptional co-activator cAMP-response element-binding protein (CREB)-binding protein (CBP)
+Syndecan-2 exerts antifibrotic effects by promoting caveolin-1-mediated transforming growth factor-b receptor I internalization and inhibiting transforming growth factor-b1 signaling
+Lower expression of platelet derived growth factor is associated with better overall survival rate of patients with idiopathic nonspecific interstitial pneumonia
+TGF-beta 1, but not TGF-beta 2 or TGF-beta 3, is differentially present in epithelial cells of advanced pulmonary fibrosis: an immunohistochemical study
+Induction of epithelial-mesenchymal transition in alveolar epithelial cells by transforming growth factor-beta1: potential role in idiopathic pulmonary fibrosis
+Significance of elevated procollagen-III-peptide and transforming growth factor-beta levels of bronchoalveolar lavage fluids from idiopathic pulmonary fibrosis patients
+The latent form of TGFbeta(1) is induced by TNFalpha through an ERK specific pathway and is activated by asbestos-derived reactive oxygen species in vitro and in vivo
+Human placental mesenchymal stem cells of fetal origins-alleviated inflammation and fibrosis by attenuating MyD88 signaling in bleomycin-induced pulmonary fibrosis mice
+Fibroblasts from idiopathic pulmonary fibrosis and normal lungs differ in growth rate, apoptosis, and tissue inhibitor of metalloproteinases expression
+Phosphatase and tensin homolog deleted on chromosome 10 contributes to phenotype transformation of fibroblasts in idiopathic pulmonary fibrosis via multiple pathways
+Transforming growth factor-b1 downregulates vascular endothelial growth factor-D expression in human lung fibroblasts via the Jun NH2-terminal kinase signaling pathway
+The anti-fibrotic effect of inhibition of TGFb-ALK5 signalling in experimental pulmonary fibrosis in mice is attenuated in the presence of concurrent y-herpesvirus infection
+Transforming growth factor-b inhibits IQ motif containing guanosine triphosphatase activating protein  1 expression in lung fibroblasts via the nuclear factor-kB signaling pathway
+De-ubiquitinating enzyme, USP11, promotes transforming growth factor b-1 signaling through stabilization of transforming growth factor b receptor II
+Aortic carboxypeptidase-like protein (ACLP) enhances lung myofibroblast differentiation through transforming growth factor b receptor-dependent and -independent pathways
+Insulin-like growth factor binding proteins 3 and 5 are overexpressed in idiopathic pulmonary fibrosis and contribute to extracellular matrix deposition
+Bile acids induce activation of alveolar epithelial cells and lung fibroblasts through farnesoid X receptor-dependent and independent pathways
+Fibroblastic foci, covered with alveolar epithelia exhibiting epithelial-mesenchymal transition, destroy alveolar septa by disrupting blood flow in idiopathic pulmonary fibrosis
+Connective tissue growth factor expression and induction by transforming growth factor-beta is abrogated by simvastatin via a Rho signaling mechanism
+Sorafenib ameliorates bleomycin-induced pulmonary fibrosis: potential roles in the inhibition of epithelial-mesenchymal transition and fibroblast activation
+NOX4/NADPH oxidase expression is increased in pulmonary fibroblasts from patients with idiopathic pulmonary fibrosis and mediates TGFbeta1-induced fibroblast differentiation into myofibroblasts
+Idiopathic pulmonary fibrosis in relation to gene polymorphisms of transforming growth factor-b1 and plasminogen activator inhibitor 1
+Comparative study of transforming growth factor-b signalling and regulatory molecules in human and canine idiopathic pulmonary fibrosis
+Microsatellite instability in transforming growth factor-beta 1 type II receptor gene in alveolar lining epithelial cells of idiopathic pulmonary fibrosis
+Prostaglandin F(2alpha) receptor signaling facilitates bleomycin-induced pulmonary fibrosis independently of transforming growth factor-beta
+Sphingosine-1-phosphate is increased in patients with idiopathic pulmonary fibrosis and mediates epithelial to mesenchymal transition
+Evaluation of permeability alteration and epithelial-mesenchymal transition induced by transforming growth factor-b1 in A549, NCI-H441, and Calu-3 cells: Development of an in vitro model of respiratory epithelial cells in idiopathic pulmonary fibrosis
+TGF-b1 stimulates HDAC4 nucleus-to-cytoplasm translocation and NADPH oxidase 4-derived reactive oxygen species in normal human lung fibroblasts
+Lactic acid is elevated in idiopathic pulmonary fibrosis and induces myofibroblast differentiation via pH-dependent activation of transforming growth factor-b
+Differential mRNA expression of insulin-like growth factor-1 splice variants in patients with idiopathic pulmonary fibrosis and pulmonary sarcoidosis
+The hedgehog system machinery controls transforming growth factor-b-dependent myofibroblastic differentiation in humans: involvement in idiopathic pulmonary fibrosis
+Reactive oxygen species are required for maintenance and differentiation of primary lung fibroblasts in idiopathic pulmonary fibrosis
+Are mast cells instrumental for fibrotic diseases? Idiopathic pulmonary fibrosis (IPF) is a fatal lung disorder of unknown etiology characterized by accumulation of lung fibroblasts and extracellular matrix deposition, ultimately leading to compromised tissue architecture and lung function capacity
+The role of halofuginone in fibrosis: more to be explored? Fibrosis, which can be defined as an abnormal or excessive accumulation of extracellular matrix (ECM), particularly fibrillar collagens, is a key driver of progressive organ dysfunction in many inflammatory and metabolic diseases, including idiopathic pulmonary fibrosis (IPF), cirrhosis, nephropathy, and oral submucous fibrosis (OSF)
+CCN5 overexpression inhibits profibrotic phenotypes via the PI3K/Akt signaling pathway in lung fibroblasts isolated from patients with idiopathic pulmonary fibrosis and in an in vivo model of lung fibrosis
+Mast cell chymase: an indispensable instrument in the pathological symphony of idiopathic pulmonary fibrosis? Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive and fatal lung disease with no known etiology and treatment options
+The Epstein-Barr virus latent membrane protein 1 and transforming growth factor--b1 synergistically induce epithelial--mesenchymal transition in lung epithelial cells
+PI3K p110y overexpression in idiopathic pulmonary fibrosis lung tissue and fibroblast cells: in vitro effects of its inhibition
+Rapamycin regulates connective tissue growth factor expression of lung epithelial cells via phosphoinositide 3-kinase
+Proliferation of pulmonary interstitial fibroblasts is mediated by transforming growth factor-beta1-induced release of extracellular fibroblast growth factor-2 and phosphorylation of p38 MAPK and JNK
+Secreted protein acidic and rich in cysteine (SPARC) is upregulated by transforming growth factor (TGF)-b and is required for TGF-b-induced hydrogen peroxide production in fibroblasts
+Semaphorin 7a+ regulatory T cells are associated with progressive idiopathic pulmonary fibrosis and are implicated in transforming growth factor-b1-induced pulmonary fibrosis
+Arsenic trioxide inhibits transforming growth factor-b1-induced fibroblast to myofibroblast differentiation in vitro and bleomycin induced lung fibrosis in vivo

+Resveratrol inhibits transforming growth factor-b-induced proliferation and differentiation of ex vivo human lung fibroblasts into myofibroblasts through <g>ERK</g>/<g>Akt</g> <u>inhibition </u>and <g>PTEN</g> restoration
+Signaling pathways in the epithelial origins of <d>pulmonary fibrosis</d>
+Raised serum levels of <g>IGFBP-1</g> and <g>IGFBP-2</g> in <d>idiopathic pulmonary fibrosis</d>
+Methylation-mediated <g>BMPER</g> <e>expression </e>in fibroblast activation in vitro and lung <d>fibrosis</d> in mice in vivo
+<d>Fibrosis</d> of two: Epithelial cell-fibroblast interactions in <d>pulmonary fibrosis</d>
+Arsenic trioxide inhibits transforming growth factor-b1-induced <d>fibroblast to myofibroblast differentiation</d> in vitro and bleomycin induced lung <d>fibrosis</d> in vivo
+microRNA-1343 attenuates pathways of <d>fibrosis</d> by targeting the <g>TGF-b</g> receptors
+<g>Matrix metalloproteinase (MMP)-19</g> <u>deficient </u>fibroblasts display a profibrotic phenotype
+Comparative study of transforming growth factor-b signalling and regulatory molecules in human and canine <d>idiopathic pulmonary fibrosis</d>
+Genetic polymorphism in <g>matrix metalloproteinase-9</g> and <g>transforming growth factor-b1</g> and susceptibility to <d>combined pulmonary fibrosis</d> and <d>emphysema</d> in  a Chinese population
+Transforming growth factor-b1 downregulates <g>vascular endothelial growth factor</g>-D expression in human lung fibroblasts via the <g>Jun NH2-terminal kinase</g> signaling pathway
+<g>Ubiquitin carboxyl-terminal hydrolase-L5</g> promotes <g>TGFb-1</g> signaling by de-ubiquitinating and stabilizing <g>Smad2</g>/<g>Smad3</g> in <d>pulmonary fibrosis</d>
+Effect of pirfenidone on proliferation, <g>TGF-b</g>-induced myofibroblast differentiation and fibrogenic activity of primary human lung fibroblasts
+[Quantifying plasma levels of <g>transforming growth factor beta1</g> in <d>idiopathic pulmonary fibrosis</d>]
+The matricellular protein <g>CCN1</g> enhances <g>TGF-b1</g>/<g>SMAD3</g>-dependent profibrotic signaling in fibroblasts and contributes to fibrogenic responses to <d>lung injury</d>
+Bleomycin in the setting of <d>lung fibrosis</d> induction: From biological mechanisms to counteractions
+Alveolar epithelial cell injury with Epstein-Barr virus <v>upregulates </v><g>TGFbeta1</g> <e>expression.</e> <d>Idiopathic pulmonary fibrosis</d> (<d>IPF</d>) is a refractory and lethal <d>interstitial lung disease</d> characterized by alveolar epithelial cells apoptosis, fibroblast proliferation, and ECM protein deposition
+Single-cell RNA sequencing identifies diverse roles of epithelial cells in <d>idiopathic pulmonary fibrosis</d>
+Data on <g>CUX1</g> isoforms in <d>idiopathic pulmonary fibrosis lung</d> and <d>systemic sclerosis</d> skin tissue sections
+<d>Idiopathic Pulmonary Fibrosis</d>: Aging, Mitochondrial Dysfunction, and Cellular Bioenergetics
+Genetic partitioning of <g>interleukin-6</g> signalling in mice dissociates <g>Stat3</g> from <g>Smad3</g>-mediated <d>lung fibrosis</d>
+Reactive oxygen species are required for maintenance and differentiation of primary lung fibroblasts in <d>idiopathic pulmonary fibrosis</d>
+Oxidant-antioxidant imbalance as a potential contributor to the progression of human <d>pulmonary fibrosis</d>
+Increased production and immunohistochemical localization of <g>transforming growth factor-beta</g> in <d>idiopathic pulmonary fibrosis</d>
+Type V collagen induced tolerance suppresses collagen deposition, <g>TGF-b</g> and associated transcripts in <d>pulmonary fibrosis</d>
+Preventive and therapeutic effects of thymosin b4 N-terminal fragment Ac-SDKP in the bleomycin model of pulmonary fibrosis
+FAK-related nonkinase is a multifunctional negative regulator of <d>pulmonary fibrosis</d>
+Nitrated fatty acids reverse <d>pulmonary fibrosis</d> by dedifferentiating myofibroblasts and promoting collagen uptake by alveolar macrophages
+Cytokine profiles in <d>idiopathic pulmonary fibrosis</d> suggest an important role for <g>TGF-beta</g> and <g>IL-10</g>
+Sphingosine-1-phosphate is increased in patients with <d>idiopathic pulmonary fibrosis</d> and mediates epithelial to mesenchymal transition
+Leucine-rich a-2 glycoprotein promotes <d>lung fibrosis</d> by modulating <g>TGF-b</g> signaling in fibroblasts
+Sustained <g>PI3K</g>
+Pirfenidone <u>inhibits </u>the <e>expression </e>of <g>HSP47</g> in <g>TGF-beta1</g>-stimulated human lung fibroblasts
+Age-driven developmental drift in the pathogenesis of <d>idiopathic pulmonary fibrosis</d>
+Endogenous <g>Semaphorin-7A</g>
+Progressive transforming growth factor beta1-induced <d>lung fibrosis</d> is blocked by an orally active <g>ALK5</g> kinase <u>inhibitor.</u> <d>Pulmonary fibrosis</d> is characterized by chronic scar formation and deposition of extracellular matrix, resulting in <d>impaired lung function</d> and <d>respiratory failure</d>
+Compromised peroxisomes in <d>idiopathic pulmonary fibrosis</d>, a vicious cycle inducing a higher fibrotic response via <g>TGF-b</g> signaling
+Inhibition of NF-kappaB signaling reduces virus load and gammaherpesvirus-induced <d>pulmonary fibrosis</d>
+Intrinsic defence capacity and therapeutic potential of natriuretic peptides in <d>pulmonary hypertension</d> associated with <d>lung fibrosis</d>
+Possible involvement of pirfenidone metabolites in the antifibrotic action of a therapy for <d>idiopathic pulmonary fibrosis</d>
+BAL cytokine profile in different <d>interstitial lung diseases</d>: a focus on <d>systemic sclerosis</d>
+Matrix metalloproteinase 3 is a mediator of <d>pulmonary fibrosis</d>
+<u>Inhibition </u>of <g>PHGDH</g>
+Bleomycin induces molecular changes directly relevant to <d>idiopathic pulmonary fibrosis</d>: a model for "active" disease
+Lysyl oxidases <r>regulate </r>fibrillar collagen remodelling in idiopathic pulmonary fibrosis
+Discovery and validation of extracellular/circulating microRNAs during <d>idiopathic pulmonary fibrosis disease</d> progression
+Recombinant human <g>serum amyloid P</g> in healthy volunteers and patients with <d>pulmonary fibrosis</d>
+<g>Transforming Growth Factor Beta 1</g>

+Diagnostic Values For Club Cell Secretory Protein (CC16) in Serum of Patients of Combined Pulmonary Fibrosis and Emphysema
+Epigenetic Regulation of Caveolin-1 Gene Expression in Lung Fibroblasts
+Targeting of Discoidin Domain Receptor 2 (DDR2) Prevents Myofibroblast Activation and Neovessel Formation During Pulmonary Fibrosis
+Epithelial Cell Mitochondrial Dysfunction and PINK1 Are Induced by Transforming Growth Factor- Beta1 in Pulmonary Fibrosis
+The lncRNA H19 Mediates Pulmonary Fibrosis by Regulating the miR-196a/COL1A1 Axis
+Inhibition of PHGDH Attenuates Bleomycin-induced Pulmonary Fibrosis
+Transforming Growth Factor Beta 1 Activation, Storage, and Signaling Pathways in Idiopathic Pulmonary Fibrosis in Dogs
+Developmental Reprogramming in Mesenchymal Stromal Cells of Human Subjects with Idiopathic Pulmonary Fibrosis
+Autoimmunity to Vimentin Is Associated with Outcomes of Patients with Idiopathic Pulmonary Fibrosis
+Amplification of TGFb Induced ITGB6 Gene Transcription May Promote Pulmonary Fibrosis
+Macrophage Bone Morphogenic Protein Receptor 2 (BMPR2) depletion in Idiopathic Pulmonary Fibrosis (IPF) and Group III Pulmonary Hypertension
+Resveratrol-Mediated Repression and Reversion of Prostatic Myofibroblast Phenoconversion
+Sunitinib, a Small-Molecule Kinase Inhibitor, Attenuates Bleomycin-Induced Pulmonary Fibrosis in Mice
+Differing Expression of Cytokines and Tumor Markers in Combined Pulmonary Fibrosis and Emphysema Compared to Emphysema and Pulmonary Fibrosis
+miR-18a-5p Inhibits Sub-pleural Pulmonary Fibrosis by Targeting TGF-b Receptor II
+Hyper-responsiveness of IPF/UIP fibroblasts: interplay between TGFbeta1, IL-13 and CCL2
+Protective Effect of Ginsenoside Rg1 on Bleomycin-Induced Pulmonary Fibrosis in Rats: Involvement of Caveolin-1 and TGF-b1 Signal Pathway
+Mesenchymal Stem Cells Correct Inappropriate Epithelial-mesenchyme Relation in Pulmonary Fibrosis Using Stanniocalcin-1
+Chop Deficiency Protects Mice Against Bleomycin-induced Pulmonary Fibrosis by Attenuating M2 Macrophage Production
+Transforming Growth Factor (TGF)-b Promotes de Novo Serine Synthesis for Collagen Production
+Two-Way Conversion between Lipogenic and Myogenic Fibroblastic Phenotypes Marks the Progression and Resolution of Lung Fibrosis
+Normal Human Lung Epithelial Cells Inhibit Transforming Growth Factor-b Induced Myofibroblast Differentiation via Prostaglandin E2
+Inhibition of the KCa3.1 Channel Alleviates Established Pulmonary Fibrosis in a Large Animal Model
+AKT2 Regulates Pulmonary Inflammation and Fibrosis via Modulating Macrophage Activation
+Novel Mechanisms for the Antifibrotic Action of Nintedanib
+Determining the True Selectivity Profile of av Integrin Ligands Using Radioligand Binding: Applying an Old Solution to a New Problem
+Increased Galectin-9 Concentration and Number of CD4+Foxp3high+Cells in Bronchoalveolar Lavage Fluid of Patients with Cryptogenic Organizing Pneumonia
+Reduced Ets Domain-containing Protein Elk1 Promotes Pulmonary Fibrosis via Increased Integrin avb6 Expression
+Expression of RXFP1 Is Decreased in Idiopathic Pulmonary Fibrosis
+The Anti-fibrotic Effects and Mechanisms of MicroRNA-486-5p in Pulmonary Fibrosis
+MAP3K19 Is a Novel Regulator of TGF-b Signaling That Impacts Bleomycin-Induced Lung Injury and Pulmonary Fibrosis
+Dysregulated Collagen Homeostasis by Matrix Stiffening and TGF-b1 in Fibroblasts from Idiopathic Pulmonary Fibrosis Patients: Role of FAK/Akt
+Sustained Activation of Toll-Like Receptor 9 Induces an Invasive Phenotype in Lung Fibroblasts: Possible Implications in Idiopathic Pulmonary Fibrosis
+Regulation of 26S Proteasome Activity in Pulmonary Fibrosis
+Membrane-anchored Serine Protease Matriptase Is a Trigger of Pulmonary Fibrogenesis
+The Role of PPARs in Lung Fibrosis
+Lipoxin A4 Attenuates Constitutive and TGF-b1-Dependent Profibrotic Activity in Human Lung Myofibroblasts
+Cigarette Smoke Enhances the Expression of Profibrotic Molecules in Alveolar Epithelial Cells
+Idiopathic Pulmonary Fibrosis: Aging, Mitochondrial Dysfunction, and Cellular Bioenergetics
+CXCL9 Regulates TGF-b1-Induced Epithelial to Mesenchymal Transition in Human Alveolar Epithelial Cells
+Anti-fibrotic Role of aB-crystallin Inhibition in Pleural and Subpleural Fibrosis
+Expression of WNT5A in Idiopathic Pulmonary Fibrosis and Its Control by TGF-b and WNT7B in Human Lung Fibroblasts

+<r>Regulation </r>of human lung fibroblast <g>C1q</g>-receptors by <g>transforming growth factor-beta</g> and <g>tumor necrosis factor-alpha</g>
+High levels of <g>IL-6</g> and <g>IL-8</g> characterize early-on <d>idiopathic pulmonary fibrosis</d> acute exacerbations
+Regulation of the effects of <g>TGF-beta 1</g> by activation of latent <g>TGF-beta 1</g> and differential <e>expression </e>of <g>TGF-beta receptors (T beta R-I and T beta R-II</g>) in <d>idiopathic pulmonary fibrosis</d>
+Peripheral depletion of NK cells and imbalance of the Treg/Th17 axis in <d>idiopathic pulmonary fibrosis</d> patients
+<g>miR-9-5p</g> suppresses pro-fibrogenic transformation of fibroblasts and prevents organ <d>fibrosis</d> by targeting <g>NOX4</g> and <g>TGFBR2</g>
+Profibrotic role of <g>WNT10A</g> via <g>TGF-b</g> signaling in <d>idiopathic pulmonary fibrosis</d>
+Microarray profiling reveals <u>suppressed </u>interferon stimulated gene program in fibroblasts from scleroderma-associated <d>interstitial lung disease</d>
+<g>IL-4</g> polymorphisms, HRCT score and lung tissue markers in <d>idiopathic pulmonary fibrosis</d>
+Herpes <d>virus infection</d> is associated with vascular remodeling and <d>pulmonary hypertension</d> in <d>idiopathic pulmonary fibrosis</d>
+Microencapsulation of lefty-secreting engineered cells for <d>pulmonary fibrosis</d> therapy in mice
+Targeting of <g>Discoidin Domain Receptor 2</g> (<g>DDR2</g>) Prevents Myofibroblast Activation and Neovessel Formation During <d>Pulmonary Fibrosis</d>
+Role of <g>protease-activated receptor-2</g> in <d>idiopathic pulmonary fibrosis</d>
+<g>Chop</g>
+<g>VCAM-1</g> is a <g>TGF-b1</g> inducible gene <v>upregulated </v>in <d>idiopathic pulmonary fibrosis</d>
+<u>Suppression </u>of <g>plasminogen activator inhibitor-1</g> by RNA interference attenuates <d>pulmonary fibrosis</d>
+<g>JAK2</g> mediates <d>lung fibrosis</d>, <d>pulmonary vascular remodelling</d> and <d>hypertension</d> in <d>idiopathic pulmonary fibrosis</d>: an experimental study
+Melatonin attenuates <g>TGFb1</g>-induced epithelial-mesenchymal transition in lung alveolar epithelial cells
+<r>Deregulation </r>of selective autophagy during aging and <d>pulmonary fibrosis</d>: the role of <g>TGFb1</g>
+<g>RhoA</g> signaling <r>modulates </r><g>cyclin D1</g> <e>expression </e>in human lung fibroblasts; implications for <d>idiopathic pulmonary fibrosis</d>
+Inhibitory effect of <g>receptor for advanced glycation end products</g> (<g>RAGE</g>) on the <g>TGF-b</g>-induced alveolar epithelial to mesenchymal transition
+<e>Effects </e>of the <d>tumor</d> suppressor <g>PTEN</g> on the pathogenesis of <d>idiopathic pulmonary fibrosis</d> in Chinese patients
+Triptolide suppresses paraquat induced <d>idiopathic pulmonary fibrosis</d> by inhibiting <g>TGFB1</g>-dependent epithelial mesenchymal transition
+<g>Endothelin-1</g> <v>induces </v>alveolar epithelial-mesenchymal transition through endothelin type A receptor-<v>mediated </v><e>production </e>of <g>TGF-beta1</g>
+<g>p63</g> - Key molecule in the early phase of <d>epithelial abnormality</d> in <d>idiopathic pulmonary fibrosis</d>
+The hedgehog system machinery controls transforming growth factor-b-dependent myofibroblastic differentiation in humans: involvement in <d>idiopathic pulmonary fibrosis</d>
+<v>Upregulation </v>of <g>activin</g>-B and <g>follistatin</g> in <d>pulmonary fibrosis</d> - a translational study using human biopsies and a specific inhibitor in mouse <d>fibrosis</d> models
+<u>Inhibition </u>of the <g>KCa3.1</g>
+Current and novel drug therapies for <d>idiopathic pulmonary fibrosis</d>
+<g>Protease activated receptor-1</g> regulates macrophage-mediated cellular senescence: a risk for <d>idiopathic pulmonary fibrosis</d>
+Sphingolipids in <d>pulmonary fibrosis</d>
+Sirtuin 7 is decreased in pulmonary fibrosis and regulates the fibrotic phenotype of lung fibroblasts
+Pirfenidone for the treatment of <d>idiopathic pulmonary fibrosis</d>
+The anti-fibrotic effect of <u>inhibition </u>of <g>TGFb</g>-<g>ALK5</g> signalling in experimental <d>pulmonary fibrosis</d> in mice is attenuated in the presence of concurrent y-herpesvirus infection
+Angiotensin-<g>TGF-beta</g> 1 crosstalk in human <d>idiopathic pulmonary fibrosis</d>: autocrine mechanisms in myofibroblasts and macrophages
+SNAI transcription factors mediate epithelial-mesenchymal transition in <d>lung fibrosis</d>
+<g>MiR-338</g>* suppresses fibrotic pathogenesis in <d>pulmonary fibrosis</d> through targeting <g>LPA1</g>
+BALF N-acetylglucosaminidase and <g>beta-galactosidase</g> activities in <d>idiopathic pulmonary fibrosis</d>
+<g>Th1</g>/Th2 cytokine gene polymorphisms in patients with <d>idiopathic pulmonary fibrosis</d>
+Transforming Growth Factor (TGF)-b Promotes de Novo Serine Synthesis for Collagen <e>Production.</e> <g>TGF-b</g> <v>promotes </v>excessive collagen deposition in <d>fibrotic diseases</d> such as <d>idiopathic pulmonary fibrosis</d> (<d>IPF</d>)
+The Epstein-Barr virus latent membrane protein 1 and transforming growth factor--b1 synergistically induce epithelial--mesenchymal transition in lung epithelial cells
+<g>Transcription factor GATA-6</g> is <e>expressed </e>in quiescent myofibroblasts in <d>idiopathic pulmonary fibrosis</d>
+Epithelial-to-mesenchymal transition and its role in <g>EGFR</g>-mutant <d>lung adenocarcinoma</d> and <d>idiopathic pulmonary fibrosis</d>

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+Tissue inhibitor of metalloproteinase-3 is up-regulated by transforming growth factor-beta1 in vitro and expressed in fibroblastic foci in vivo in idiopathic pulmonary fibrosis
+Upregulation of alveolar levels of activin B, but not activin A, in lungs of west highland white terriers with idiopathic pulmonary fibrosis and diffuse alveolar damage
+Progressive transforming growth factor beta1-induced lung fibrosis is blocked by an orally active ALK5 kinase inhibitor
+Upregulation of activin-B and follistatin in pulmonary fibrosis - a translational study using human biopsies and a specific inhibitor in mouse fibrosis models
+Resveratrol inhibits transforming growth factor-b-induced proliferation and differentiation of ex vivo human lung fibroblasts into myofibroblasts through ERK/Akt inhibition and PTEN restoration
+Interactions between b-catenin and transforming growth factor-b signaling pathways mediate epithelial-mesenchymal transition and are dependent on the transcriptional co-activator cAMP-response element-binding protein (CREB)-binding protein (CBP)
+Syndecan-2 exerts antifibrotic effects by promoting caveolin-1-mediated transforming growth factor-b receptor I internalization and inhibiting transforming growth factor-b1 signaling
+Lower expression of platelet derived growth factor is associated with better overall survival rate of patients with idiopathic nonspecific interstitial pneumonia
+TGF-beta 1, but not TGF-beta 2 or TGF-beta 3, is differentially present in epithelial cells of advanced pulmonary fibrosis: an immunohistochemical study
+Induction of epithelial-mesenchymal transition in alveolar epithelial cells by transforming growth factor-beta1: potential role in idiopathic pulmonary fibrosis
+Significance of elevated procollagen-III-peptide and transforming growth factor-beta levels of bronchoalveolar lavage fluids from idiopathic pulmonary fibrosis patients
+The latent form of TGFbeta(1) is induced by TNFalpha through an ERK specific pathway and is activated by asbestos-derived reactive oxygen species in vitro and in vivo
+Human placental mesenchymal stem cells of fetal origins-alleviated inflammation and fibrosis by attenuating MyD88 signaling in bleomycin-induced pulmonary fibrosis mice
+Fibroblasts from idiopathic pulmonary fibrosis and normal lungs differ in growth rate, apoptosis, and tissue inhibitor of metalloproteinases expression
+Phosphatase and tensin homolog deleted on chromosome 10 contributes to phenotype transformation of fibroblasts in idiopathic pulmonary fibrosis via multiple pathways
+Transforming growth factor-b1 downregulates vascular endothelial growth factor-D expression in human lung fibroblasts via the Jun NH2-terminal kinase signaling pathway
+The anti-fibrotic effect of inhibition of TGFb-ALK5 signalling in experimental pulmonary fibrosis in mice is attenuated in the presence of concurrent y-herpesvirus infection
+Transforming growth factor-b inhibits IQ motif containing guanosine triphosphatase activating protein  1 expression in lung fibroblasts via the nuclear factor-kB signaling pathway
+De-ubiquitinating enzyme, USP11, promotes transforming growth factor b-1 signaling through stabilization of transforming growth factor b receptor II
+Aortic carboxypeptidase-like protein (ACLP) enhances lung myofibroblast differentiation through transforming growth factor b receptor-dependent and -independent pathways
+Insulin-like growth factor binding proteins 3 and 5 are overexpressed in idiopathic pulmonary fibrosis and contribute to extracellular matrix deposition
+Bile acids induce activation of alveolar epithelial cells and lung fibroblasts through farnesoid X receptor-dependent and independent pathways
+Fibroblastic foci, covered with alveolar epithelia exhibiting epithelial-mesenchymal transition, destroy alveolar septa by disrupting blood flow in idiopathic pulmonary fibrosis
+Connective tissue growth factor expression and induction by transforming growth factor-beta is abrogated by simvastatin via a Rho signaling mechanism
+Sorafenib ameliorates bleomycin-induced pulmonary fibrosis: potential roles in the inhibition of epithelial-mesenchymal transition and fibroblast activation
+NOX4/NADPH oxidase expression is increased in pulmonary fibroblasts from patients with idiopathic pulmonary fibrosis and mediates TGFbeta1-induced fibroblast differentiation into myofibroblasts
+Idiopathic pulmonary fibrosis in relation to gene polymorphisms of transforming growth factor-b1 and plasminogen activator inhibitor 1
+Comparative study of transforming growth factor-b signalling and regulatory molecules in human and canine idiopathic pulmonary fibrosis
+Microsatellite instability in transforming growth factor-beta 1 type II receptor gene in alveolar lining epithelial cells of idiopathic pulmonary fibrosis
+Prostaglandin F(2alpha) receptor signaling facilitates bleomycin-induced pulmonary fibrosis independently of transforming growth factor-beta
+Sphingosine-1-phosphate is increased in patients with idiopathic pulmonary fibrosis and mediates epithelial to mesenchymal transition
+Evaluation of permeability alteration and epithelial-mesenchymal transition induced by transforming growth factor-b1 in A549, NCI-H441, and Calu-3 cells: Development of an in vitro model of respiratory epithelial cells in idiopathic pulmonary fibrosis
+TGF-b1 stimulates HDAC4 nucleus-to-cytoplasm translocation and NADPH oxidase 4-derived reactive oxygen species in normal human lung fibroblasts
+Lactic acid is elevated in idiopathic pulmonary fibrosis and induces myofibroblast differentiation via pH-dependent activation of transforming growth factor-b
+Differential mRNA expression of insulin-like growth factor-1 splice variants in patients with idiopathic pulmonary fibrosis and pulmonary sarcoidosis
+The hedgehog system machinery controls transforming growth factor-b-dependent myofibroblastic differentiation in humans: involvement in idiopathic pulmonary fibrosis
+Reactive oxygen species are required for maintenance and differentiation of primary lung fibroblasts in idiopathic pulmonary fibrosis
+Are mast cells instrumental for fibrotic diseases? Idiopathic pulmonary fibrosis (IPF) is a fatal lung disorder of unknown etiology characterized by accumulation of lung fibroblasts and extracellular matrix deposition, ultimately leading to compromised tissue architecture and lung function capacity
+The role of halofuginone in fibrosis: more to be explored? Fibrosis, which can be defined as an abnormal or excessive accumulation of extracellular matrix (ECM), particularly fibrillar collagens, is a key driver of progressive organ dysfunction in many inflammatory and metabolic diseases, including idiopathic pulmonary fibrosis (IPF), cirrhosis, nephropathy, and oral submucous fibrosis (OSF)
+CCN5 overexpression inhibits profibrotic phenotypes via the PI3K/Akt signaling pathway in lung fibroblasts isolated from patients with idiopathic pulmonary fibrosis and in an in vivo model of lung fibrosis
+Mast cell chymase: an indispensable instrument in the pathological symphony of idiopathic pulmonary fibrosis? Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive and fatal lung disease with no known etiology and treatment options
+The Epstein-Barr virus latent membrane protein 1 and transforming growth factor--b1 synergistically induce epithelial--mesenchymal transition in lung epithelial cells
+PI3K p110y overexpression in idiopathic pulmonary fibrosis lung tissue and fibroblast cells: in vitro effects of its inhibition
+Rapamycin regulates connective tissue growth factor expression of lung epithelial cells via phosphoinositide 3-kinase
+Proliferation of pulmonary interstitial fibroblasts is mediated by transforming growth factor-beta1-induced release of extracellular fibroblast growth factor-2 and phosphorylation of p38 MAPK and JNK
+Secreted protein acidic and rich in cysteine (SPARC) is upregulated by transforming growth factor (TGF)-b and is required for TGF-b-induced hydrogen peroxide production in fibroblasts
+Semaphorin 7a+ regulatory T cells are associated with progressive idiopathic pulmonary fibrosis and are implicated in transforming growth factor-b1-induced pulmonary fibrosis
+Arsenic trioxide inhibits transforming growth factor-b1-induced fibroblast to myofibroblast differentiation in vitro and bleomycin induced lung fibrosis in vivo

+Resveratrol inhibits transforming growth factor-b-induced proliferation and differentiation of ex vivo human lung fibroblasts into myofibroblasts through <g>ERK</g>/<g>Akt</g> <u>inhibition </u>and <g>PTEN</g> restoration
+Signaling pathways in the epithelial origins of <d>pulmonary fibrosis</d>
+Raised serum levels of <g>IGFBP-1</g> and <g>IGFBP-2</g> in <d>idiopathic pulmonary fibrosis</d>
+Methylation-mediated <g>BMPER</g> <e>expression </e>in fibroblast activation in vitro and lung <d>fibrosis</d> in mice in vivo
+<d>Fibrosis</d> of two: Epithelial cell-fibroblast interactions in <d>pulmonary fibrosis</d>
+Arsenic trioxide inhibits transforming growth factor-b1-induced <d>fibroblast to myofibroblast differentiation</d> in vitro and bleomycin induced lung <d>fibrosis</d> in vivo
+microRNA-1343 attenuates pathways of <d>fibrosis</d> by targeting the <g>TGF-b</g> receptors
+<g>Matrix metalloproteinase (MMP)-19</g> <u>deficient </u>fibroblasts display a profibrotic phenotype
+Comparative study of transforming growth factor-b signalling and regulatory molecules in human and canine <d>idiopathic pulmonary fibrosis</d>
+Genetic polymorphism in <g>matrix metalloproteinase-9</g> and <g>transforming growth factor-b1</g> and susceptibility to <d>combined pulmonary fibrosis</d> and <d>emphysema</d> in  a Chinese population
+Transforming growth factor-b1 downregulates <g>vascular endothelial growth factor</g>-D expression in human lung fibroblasts via the <g>Jun NH2-terminal kinase</g> signaling pathway
+<g>Ubiquitin carboxyl-terminal hydrolase-L5</g> promotes <g>TGFb-1</g> signaling by de-ubiquitinating and stabilizing <g>Smad2</g>/<g>Smad3</g> in <d>pulmonary fibrosis</d>
+Effect of pirfenidone on proliferation, <g>TGF-b</g>-induced myofibroblast differentiation and fibrogenic activity of primary human lung fibroblasts
+[Quantifying plasma levels of <g>transforming growth factor beta1</g> in <d>idiopathic pulmonary fibrosis</d>]
+The matricellular protein <g>CCN1</g> enhances <g>TGF-b1</g>/<g>SMAD3</g>-dependent profibrotic signaling in fibroblasts and contributes to fibrogenic responses to <d>lung injury</d>
+Bleomycin in the setting of <d>lung fibrosis</d> induction: From biological mechanisms to counteractions
+Alveolar epithelial cell injury with Epstein-Barr virus <v>upregulates </v><g>TGFbeta1</g> <e>expression.</e> <d>Idiopathic pulmonary fibrosis</d> (<d>IPF</d>) is a refractory and lethal <d>interstitial lung disease</d> characterized by alveolar epithelial cells apoptosis, fibroblast proliferation, and ECM protein deposition
+Single-cell RNA sequencing identifies diverse roles of epithelial cells in <d>idiopathic pulmonary fibrosis</d>
+Data on <g>CUX1</g> isoforms in <d>idiopathic pulmonary fibrosis lung</d> and <d>systemic sclerosis</d> skin tissue sections
+<d>Idiopathic Pulmonary Fibrosis</d>: Aging, Mitochondrial Dysfunction, and Cellular Bioenergetics
+Genetic partitioning of <g>interleukin-6</g> signalling in mice dissociates <g>Stat3</g> from <g>Smad3</g>-mediated <d>lung fibrosis</d>
+Reactive oxygen species are required for maintenance and differentiation of primary lung fibroblasts in <d>idiopathic pulmonary fibrosis</d>
+Oxidant-antioxidant imbalance as a potential contributor to the progression of human <d>pulmonary fibrosis</d>
+Increased production and immunohistochemical localization of <g>transforming growth factor-beta</g> in <d>idiopathic pulmonary fibrosis</d>
+Type V collagen induced tolerance suppresses collagen deposition, <g>TGF-b</g> and associated transcripts in <d>pulmonary fibrosis</d>
+Preventive and therapeutic effects of thymosin b4 N-terminal fragment Ac-SDKP in the bleomycin model of pulmonary fibrosis
+FAK-related nonkinase is a multifunctional negative regulator of <d>pulmonary fibrosis</d>
+Nitrated fatty acids reverse <d>pulmonary fibrosis</d> by dedifferentiating myofibroblasts and promoting collagen uptake by alveolar macrophages
+Cytokine profiles in <d>idiopathic pulmonary fibrosis</d> suggest an important role for <g>TGF-beta</g> and <g>IL-10</g>
+Sphingosine-1-phosphate is increased in patients with <d>idiopathic pulmonary fibrosis</d> and mediates epithelial to mesenchymal transition
+Leucine-rich a-2 glycoprotein promotes <d>lung fibrosis</d> by modulating <g>TGF-b</g> signaling in fibroblasts
+Sustained <g>PI3K</g>
+Pirfenidone <u>inhibits </u>the <e>expression </e>of <g>HSP47</g> in <g>TGF-beta1</g>-stimulated human lung fibroblasts
+Age-driven developmental drift in the pathogenesis of <d>idiopathic pulmonary fibrosis</d>
+Endogenous <g>Semaphorin-7A</g>
+Progressive transforming growth factor beta1-induced <d>lung fibrosis</d> is blocked by an orally active <g>ALK5</g> kinase <u>inhibitor.</u> <d>Pulmonary fibrosis</d> is characterized by chronic scar formation and deposition of extracellular matrix, resulting in <d>impaired lung function</d> and <d>respiratory failure</d>
+Compromised peroxisomes in <d>idiopathic pulmonary fibrosis</d>, a vicious cycle inducing a higher fibrotic response via <g>TGF-b</g> signaling
+Inhibition of NF-kappaB signaling reduces virus load and gammaherpesvirus-induced <d>pulmonary fibrosis</d>
+Intrinsic defence capacity and therapeutic potential of natriuretic peptides in <d>pulmonary hypertension</d> associated with <d>lung fibrosis</d>
+Possible involvement of pirfenidone metabolites in the antifibrotic action of a therapy for <d>idiopathic pulmonary fibrosis</d>
+BAL cytokine profile in different <d>interstitial lung diseases</d>: a focus on <d>systemic sclerosis</d>
+Matrix metalloproteinase 3 is a mediator of <d>pulmonary fibrosis</d>
+<u>Inhibition </u>of <g>PHGDH</g>
+Bleomycin induces molecular changes directly relevant to <d>idiopathic pulmonary fibrosis</d>: a model for "active" disease
+Lysyl oxidases <r>regulate </r>fibrillar collagen remodelling in idiopathic pulmonary fibrosis
+Discovery and validation of extracellular/circulating microRNAs during <d>idiopathic pulmonary fibrosis disease</d> progression
+Recombinant human <g>serum amyloid P</g> in healthy volunteers and patients with <d>pulmonary fibrosis</d>
+<g>Transforming Growth Factor Beta 1</g>

+Diagnostic Values For Club Cell Secretory Protein (CC16) in Serum of Patients of Combined Pulmonary Fibrosis and Emphysema
+Epigenetic Regulation of Caveolin-1 Gene Expression in Lung Fibroblasts
+Targeting of Discoidin Domain Receptor 2 (DDR2) Prevents Myofibroblast Activation and Neovessel Formation During Pulmonary Fibrosis
+Epithelial Cell Mitochondrial Dysfunction and PINK1 Are Induced by Transforming Growth Factor- Beta1 in Pulmonary Fibrosis
+The lncRNA H19 Mediates Pulmonary Fibrosis by Regulating the miR-196a/COL1A1 Axis
+Inhibition of PHGDH Attenuates Bleomycin-induced Pulmonary Fibrosis
+Transforming Growth Factor Beta 1 Activation, Storage, and Signaling Pathways in Idiopathic Pulmonary Fibrosis in Dogs
+Developmental Reprogramming in Mesenchymal Stromal Cells of Human Subjects with Idiopathic Pulmonary Fibrosis
+Autoimmunity to Vimentin Is Associated with Outcomes of Patients with Idiopathic Pulmonary Fibrosis
+Amplification of TGFb Induced ITGB6 Gene Transcription May Promote Pulmonary Fibrosis
+Macrophage Bone Morphogenic Protein Receptor 2 (BMPR2) depletion in Idiopathic Pulmonary Fibrosis (IPF) and Group III Pulmonary Hypertension
+Resveratrol-Mediated Repression and Reversion of Prostatic Myofibroblast Phenoconversion
+Sunitinib, a Small-Molecule Kinase Inhibitor, Attenuates Bleomycin-Induced Pulmonary Fibrosis in Mice
+Differing Expression of Cytokines and Tumor Markers in Combined Pulmonary Fibrosis and Emphysema Compared to Emphysema and Pulmonary Fibrosis
+miR-18a-5p Inhibits Sub-pleural Pulmonary Fibrosis by Targeting TGF-b Receptor II
+Hyper-responsiveness of IPF/UIP fibroblasts: interplay between TGFbeta1, IL-13 and CCL2
+Protective Effect of Ginsenoside Rg1 on Bleomycin-Induced Pulmonary Fibrosis in Rats: Involvement of Caveolin-1 and TGF-b1 Signal Pathway
+Mesenchymal Stem Cells Correct Inappropriate Epithelial-mesenchyme Relation in Pulmonary Fibrosis Using Stanniocalcin-1
+Chop Deficiency Protects Mice Against Bleomycin-induced Pulmonary Fibrosis by Attenuating M2 Macrophage Production
+Transforming Growth Factor (TGF)-b Promotes de Novo Serine Synthesis for Collagen Production
+Two-Way Conversion between Lipogenic and Myogenic Fibroblastic Phenotypes Marks the Progression and Resolution of Lung Fibrosis
+Normal Human Lung Epithelial Cells Inhibit Transforming Growth Factor-b Induced Myofibroblast Differentiation via Prostaglandin E2
+Inhibition of the KCa3.1 Channel Alleviates Established Pulmonary Fibrosis in a Large Animal Model
+AKT2 Regulates Pulmonary Inflammation and Fibrosis via Modulating Macrophage Activation
+Novel Mechanisms for the Antifibrotic Action of Nintedanib
+Determining the True Selectivity Profile of av Integrin Ligands Using Radioligand Binding: Applying an Old Solution to a New Problem
+Increased Galectin-9 Concentration and Number of CD4+Foxp3high+Cells in Bronchoalveolar Lavage Fluid of Patients with Cryptogenic Organizing Pneumonia
+Reduced Ets Domain-containing Protein Elk1 Promotes Pulmonary Fibrosis via Increased Integrin avb6 Expression
+Expression of RXFP1 Is Decreased in Idiopathic Pulmonary Fibrosis
+The Anti-fibrotic Effects and Mechanisms of MicroRNA-486-5p in Pulmonary Fibrosis
+MAP3K19 Is a Novel Regulator of TGF-b Signaling That Impacts Bleomycin-Induced Lung Injury and Pulmonary Fibrosis
+Dysregulated Collagen Homeostasis by Matrix Stiffening and TGF-b1 in Fibroblasts from Idiopathic Pulmonary Fibrosis Patients: Role of FAK/Akt
+Sustained Activation of Toll-Like Receptor 9 Induces an Invasive Phenotype in Lung Fibroblasts: Possible Implications in Idiopathic Pulmonary Fibrosis
+Regulation of 26S Proteasome Activity in Pulmonary Fibrosis
+Membrane-anchored Serine Protease Matriptase Is a Trigger of Pulmonary Fibrogenesis
+The Role of PPARs in Lung Fibrosis
+Lipoxin A4 Attenuates Constitutive and TGF-b1-Dependent Profibrotic Activity in Human Lung Myofibroblasts
+Cigarette Smoke Enhances the Expression of Profibrotic Molecules in Alveolar Epithelial Cells
+Idiopathic Pulmonary Fibrosis: Aging, Mitochondrial Dysfunction, and Cellular Bioenergetics
+CXCL9 Regulates TGF-b1-Induced Epithelial to Mesenchymal Transition in Human Alveolar Epithelial Cells
+Anti-fibrotic Role of aB-crystallin Inhibition in Pleural and Subpleural Fibrosis
+Expression of WNT5A in Idiopathic Pulmonary Fibrosis and Its Control by TGF-b and WNT7B in Human Lung Fibroblasts

+<r>Regulation </r>of human lung fibroblast <g>C1q</g>-receptors by <g>transforming growth factor-beta</g> and <g>tumor necrosis factor-alpha</g>
+High levels of <g>IL-6</g> and <g>IL-8</g> characterize early-on <d>idiopathic pulmonary fibrosis</d> acute exacerbations
+Regulation of the effects of <g>TGF-beta 1</g> by activation of latent <g>TGF-beta 1</g> and differential <e>expression </e>of <g>TGF-beta receptors (T beta R-I and T beta R-II</g>) in <d>idiopathic pulmonary fibrosis</d>
+Peripheral depletion of NK cells and imbalance of the Treg/Th17 axis in <d>idiopathic pulmonary fibrosis</d> patients
+<g>miR-9-5p</g> suppresses pro-fibrogenic transformation of fibroblasts and prevents organ <d>fibrosis</d> by targeting <g>NOX4</g> and <g>TGFBR2</g>
+Profibrotic role of <g>WNT10A</g> via <g>TGF-b</g> signaling in <d>idiopathic pulmonary fibrosis</d>
+Microarray profiling reveals <u>suppressed </u>interferon stimulated gene program in fibroblasts from scleroderma-associated <d>interstitial lung disease</d>
+<g>IL-4</g> polymorphisms, HRCT score and lung tissue markers in <d>idiopathic pulmonary fibrosis</d>
+Herpes <d>virus infection</d> is associated with vascular remodeling and <d>pulmonary hypertension</d> in <d>idiopathic pulmonary fibrosis</d>
+Microencapsulation of lefty-secreting engineered cells for <d>pulmonary fibrosis</d> therapy in mice
+Targeting of <g>Discoidin Domain Receptor 2</g> (<g>DDR2</g>) Prevents Myofibroblast Activation and Neovessel Formation During <d>Pulmonary Fibrosis</d>
+Role of <g>protease-activated receptor-2</g> in <d>idiopathic pulmonary fibrosis</d>
+<g>Chop</g>
+<g>VCAM-1</g> is a <g>TGF-b1</g> inducible gene <v>upregulated </v>in <d>idiopathic pulmonary fibrosis</d>
+<u>Suppression </u>of <g>plasminogen activator inhibitor-1</g> by RNA interference attenuates <d>pulmonary fibrosis</d>
+<g>JAK2</g> mediates <d>lung fibrosis</d>, <d>pulmonary vascular remodelling</d> and <d>hypertension</d> in <d>idiopathic pulmonary fibrosis</d>: an experimental study
+Melatonin attenuates <g>TGFb1</g>-induced epithelial-mesenchymal transition in lung alveolar epithelial cells
+<r>Deregulation </r>of selective autophagy during aging and <d>pulmonary fibrosis</d>: the role of <g>TGFb1</g>
+<g>RhoA</g> signaling <r>modulates </r><g>cyclin D1</g> <e>expression </e>in human lung fibroblasts; implications for <d>idiopathic pulmonary fibrosis</d>
+Inhibitory effect of <g>receptor for advanced glycation end products</g> (<g>RAGE</g>) on the <g>TGF-b</g>-induced alveolar epithelial to mesenchymal transition
+<e>Effects </e>of the <d>tumor</d> suppressor <g>PTEN</g> on the pathogenesis of <d>idiopathic pulmonary fibrosis</d> in Chinese patients
+Triptolide suppresses paraquat induced <d>idiopathic pulmonary fibrosis</d> by inhibiting <g>TGFB1</g>-dependent epithelial mesenchymal transition
+<g>Endothelin-1</g> <v>induces </v>alveolar epithelial-mesenchymal transition through endothelin type A receptor-<v>mediated </v><e>production </e>of <g>TGF-beta1</g>
+<g>p63</g> - Key molecule in the early phase of <d>epithelial abnormality</d> in <d>idiopathic pulmonary fibrosis</d>
+The hedgehog system machinery controls transforming growth factor-b-dependent myofibroblastic differentiation in humans: involvement in <d>idiopathic pulmonary fibrosis</d>
+<v>Upregulation </v>of <g>activin</g>-B and <g>follistatin</g> in <d>pulmonary fibrosis</d> - a translational study using human biopsies and a specific inhibitor in mouse <d>fibrosis</d> models
+<u>Inhibition </u>of the <g>KCa3.1</g>
+Current and novel drug therapies for <d>idiopathic pulmonary fibrosis</d>
+<g>Protease activated receptor-1</g> regulates macrophage-mediated cellular senescence: a risk for <d>idiopathic pulmonary fibrosis</d>
+Sphingolipids in <d>pulmonary fibrosis</d>
+Sirtuin 7 is decreased in pulmonary fibrosis and regulates the fibrotic phenotype of lung fibroblasts
+Pirfenidone for the treatment of <d>idiopathic pulmonary fibrosis</d>
+The anti-fibrotic effect of <u>inhibition </u>of <g>TGFb</g>-<g>ALK5</g> signalling in experimental <d>pulmonary fibrosis</d> in mice is attenuated in the presence of concurrent y-herpesvirus infection
+Angiotensin-<g>TGF-beta</g> 1 crosstalk in human <d>idiopathic pulmonary fibrosis</d>: autocrine mechanisms in myofibroblasts and macrophages
+SNAI transcription factors mediate epithelial-mesenchymal transition in <d>lung fibrosis</d>
+<g>MiR-338</g>* suppresses fibrotic pathogenesis in <d>pulmonary fibrosis</d> through targeting <g>LPA1</g>
+BALF N-acetylglucosaminidase and <g>beta-galactosidase</g> activities in <d>idiopathic pulmonary fibrosis</d>
+<g>Th1</g>/Th2 cytokine gene polymorphisms in patients with <d>idiopathic pulmonary fibrosis</d>
+Transforming Growth Factor (TGF)-b Promotes de Novo Serine Synthesis for Collagen <e>Production.</e> <g>TGF-b</g> <v>promotes </v>excessive collagen deposition in <d>fibrotic diseases</d> such as <d>idiopathic pulmonary fibrosis</d> (<d>IPF</d>)
+The Epstein-Barr virus latent membrane protein 1 and transforming growth factor--b1 synergistically induce epithelial--mesenchymal transition in lung epithelial cells
+<g>Transcription factor GATA-6</g> is <e>expressed </e>in quiescent myofibroblasts in <d>idiopathic pulmonary fibrosis</d>
+Epithelial-to-mesenchymal transition and its role in <g>EGFR</g>-mutant <d>lung adenocarcinoma</d> and <d>idiopathic pulmonary fibrosis</d>

+miR-26a suppresses EMT by disrupting the Lin28B/let-7d axis: potential cross-talks among miRNAs in IPF
+MiR-185/AKT and miR-29a/collagen 1a pathways are activated in IPF BAL cells
+Tubastatin ameliorates pulmonary fibrosis by targeting the TGFb-PI3K-Akt pathway
+Pirfenidone inhibits the expression of HSP47 in TGF-beta1-stimulated human lung fibroblasts
+Effects of particulate matter from straw burning on lung fibrosis in mice
+Regulation of myofibroblast differentiation by miR-424 during epithelial-to-mesenchymal transition
+Effect of substrate stiffness on pulmonary fibroblast activation by TGF-b
+Shikonin suppresses pulmonary fibroblasts proliferation and activation by regulating Akt and p38 MAPK signaling pathways
+Gremlin-mediated decrease in bone morphogenetic protein signaling promotes pulmonary fibrosis
+miR-92a regulates TGF-b1-induced WISP1 expression in pulmonary fibrosis
+Transgelin is a direct target of TGF-beta/Smad3-dependent epithelial cell migration in lung fibrosis
+Lung myofibroblasts are characterized by down-regulated cyclooxygenase-2 and its main metabolite, prostaglandin E2
+Fibrotic myofibroblasts manifest genome-wide derangements of translational control
+Rapamycin increases CCN2 expression of lung fibroblasts via phosphoinositide 3-kinase
+Genetic partitioning of interleukin-6 signalling in mice dissociates Stat3 from Smad3-mediated lung fibrosis
+Latent cytomegalovirus infection exacerbates experimental pulmonary fibrosis by activating TGF-b1
+TGF-beta driven lung fibrosis is macrophage dependent and blocked by Serum amyloid P
+Lysocardiolipin acyltransferase regulates TGF-b mediated lung fibroblast differentiation
+Suppression of plasminogen activator inhibitor-1 by RNA interference attenuates pulmonary fibrosis
+Pirfenidone exerts antifibrotic effects through inhibition of GLI transcription factors
+microRNA-1343 attenuates pathways of fibrosis by targeting the TGF-b receptors
+Effect of Renshen Pingfei Decoction, a traditional Chinese prescription, on IPF induced by Bleomycin in rats and regulation of TGF-b1/Smad3
+M2 macrophages induce EMT through the TGF-b/Smad2 signaling pathway
+miR  -221 targets HMGA2 to inhibit bleomycin  -induced pulmonary fibrosis by regulating TGF  -b1/Smad3-induced EMT
+FGF-1 reverts epithelial-mesenchymal transition induced by TGF-{beta}1 through MAPK/ERK kinase pathway
+Inhibition of HSP27 blocks fibrosis development and EMT features by promoting Snail degradation
+Human lung myofibroblast TGFb1-dependent Smad2/3 signalling is Ca(2+)-dependent and regulated by KCa3.1 K(+) channels
+The anti-fibrotic effects of microRNA-153 by targeting TGFBR-2 in pulmonary fibrosis
+MicroRNA-101 attenuates pulmonary fibrosis by inhibiting fibroblast proliferation and activation
+Ubiquitin carboxyl-terminal hydrolase-L5 promotes TGFb-1 signaling by de-ubiquitinating and stabilizing Smad2/Smad3 in pulmonary fibrosis
+Cub domain containing protein 1 (CDCP1) negatively regulates TGFb signaling and myofibroblast differentiation
+The mannose-6-phosphate analogue, PXS64, inhibits fibrosis via TGF-b1 pathway in human lung fibroblasts
+Smooth muscle alpha-actin expression and myofibroblast differentiation by TGFbeta are dependent upon MK2
+miR-323a-3p regulates lung fibrosis by targeting multiple profibrotic pathways
+Low-dose paclitaxel ameliorates pulmonary fibrosis by suppressing TGF-b1/Smad3 pathway via miR-140 upregulation
+MiR-338* targeting smoothened to inhibit pulmonary fibrosis by epithelial-mesenchymal transition
+N-acetyl-L-cysteine inhibits TGF-beta1-induced profibrotic responses in fibroblasts
+Sustained PI3K Activation exacerbates BLM-induced Lung Fibrosis via activation of pro-inflammatory and pro-fibrotic pathways
+miR-9-5p suppresses pro-fibrogenic transformation of fibroblasts and prevents organ fibrosis by targeting NOX4 and TGFBR2
+Role of integrin-mediated TGFbeta activation in the pathogenesis of pulmonary fibrosis
+Leucine-rich a-2 glycoprotein promotes lung fibrosis by modulating TGF-b signaling in fibroblasts
+MiR-541-5p regulates lung fibrosis by targeting cyclic nucleotide phosphodiesterase 1A
+Sputum biomarkers in IPF: Evidence for raised gene expression and protein level of IGFBP-2, IL-8 and MMP-7
+STAT3-mediated signaling dysregulates lung fibroblast-myofibroblast activation and differentiation in UIP/IPF
+TGF-b1 induces Fstl1 via the Smad3-c-Jun pathway in lung fibroblasts
+HSP27 regulates TGF-b mediated lung fibroblast differentiation through the Smad3 and ERK pathways
+SNAI transcription factors mediate epithelial-mesenchymal transition in lung fibrosis
+Bone morphogenetic protein-inducer tilorone identified by high-throughput screening is antifibrotic in vivo
+Endothelin-1 induces alveolar epithelial-mesenchymal transition through endothelin type A receptor-mediated production of TGF-beta1
+Lung fibrotic tenascin-C upregulation is associated with other extracellular matrix proteins and induced by TGFb1
+Abrogation of TGF-beta1-induced fibroblast-myofibroblast differentiation by histone deacetylase inhibition
+Negative regulation of myofibroblast differentiation by PTEN (Phosphatase and Tensin Homolog Deleted on chromosome 10)

+Comparison between conventional and "clinical" assessment of experimental lung <d>fibrosis</d>
+Developmental Reprogramming in Mesenchymal Stromal Cells of Human Subjects with <d>Idiopathic Pulmonary Fibrosis</d>
+<u>Inhibition </u>of <g>HSP27</g> blocks <d>fibrosis</d> development and EMT features by promoting <g>Snail</g> degradation
+<r>Effects </r>of a leukotriene B4 receptor antagonist on bleomycin-induced <d>pulmonary fibrosis</d>
+Activated human T lymphocytes inhibit <g>TGFb</g>-induced fibroblast to myofibroblast differentiation via prostaglandins D2 and E2
+The potential application of strategic released apigenin from polymeric carrier in pulmonary fibrosis
+<g>Bone morphogenetic protein-4</g> inhibitor <g>gremlin</g> is <v>overexpressed </v>in <d>idiopathic pulmonary fibrosis</d>
+The K+ channel <g>KCa3.1</g> as a novel target for <d>idiopathic pulmonary fibrosis</d>
+<g>Spiruchostatin A</g> inhibits proliferation and differentiation of fibroblasts from patients with <d>pulmonary fibrosis</d>
+Lung myofibroblasts are characterized by <u>down-regulated </u><g>cyclooxygenase-2</g> and its main metabolite, prostaglandin E2
+Cigarette Smoke Enhances the Expression of Profibrotic Molecules in Alveolar Epithelial Cells
+Effect of substrate stiffness on pulmonary fibroblast activation by <g>TGF-b</g>
+Bile acids induce activation of alveolar epithelial cells and lung fibroblasts through farnesoid X receptor-dependent and independent pathways
+Exercise <u>Reduces </u>Lung <d>Fibrosis</d>
+<e>Expression </e>of mucosa-related integrin alphaEbeta7 on <d>alveolar T</d> cells in <d>interstitial lung diseases</d>
+Fibroblasts from <d>idiopathic pulmonary fibrosis</d> and normal lungs differ in growth rate, apoptosis, and tissue <u>inhibitor </u>of metalloproteinases <e>expression.</e> <d>Idiopathic pulmonary fibrosis</d> (<d>IPF</d>) is a <d>chronic lung disorder</d> characterized by fibroblast proliferation and extracellular matrix accumulation
+<d>Idiopathic pulmonary fibrosis</d>: immunohistochemical analysis provides fresh insights into lung tissue remodelling with implications for novel prognostic markers
+Amplified canonical transforming growth factor-b signalling<i>via</i><d>heat shock</d> protein 90 in <d>pulmonary fibrosis</d>
+Long-acting human serum albumin-thioredoxin fusion protein suppresses bleomycin-induced <d>pulmonary fibrosis progression</d>
+Fibrogenic Lung Injury Induces Non-Cell-Autonomous Fibroblast Invasion
+Recent advances in molecular targets and treatment of <d>idiopathic pulmonary fibrosis</d>: focus on <g>TGFbeta</g> signaling and the myofibroblast
+<g>Bone morphogenetic protein</g>-inducer tilorone identified by high-throughput screening is antifibrotic in vivo
+Pleural mesothelial cell differentiation and invasion in <d>fibrogenic lung injury</d>
+Studies of <g>hepatocyte growth factor</g> in bronchoalveolar lavage fluid in chronic <d>interstitial lung diseases</d>
+Are mast cells instrumental for <d>fibrotic diseases</d>? <d>Idiopathic pulmonary fibrosis</d> (<d>IPF</d>) is a <d>fatal lung disorder</d> of unknown etiology characterized by accumulation of lung fibroblasts and extracellular matrix deposition, ultimately leading to compromised tissue architecture and lung function capacity
+Effects of <d>cigarette smoke extract</d> on A549 cells and human lung fibroblasts treated with <g>transforming growth factor-beta1</g> in a coculture system
+Differential mRNA expression of <g>insulin-like growth factor-1</g> splice variants in patients with <d>idiopathic pulmonary fibrosis</d> and <d>pulmonary sarcoidosis</d>
+<u>Reduced </u><g>Ets Domain-containing Protein Elk1</g>
+<g>HSP27</g> regulates <g>TGF-b</g> mediated lung fibroblast differentiation through the <g>Smad3</g> and <g>ERK</g> pathways
+<g>Transforming growth factor b1</g> (<g>TGFb1</g>)-induced CD44V6-<g>NOX4</g> signaling in pathogenesis of <d>idiopathic pulmonary fibrosis</d>
+Pleural mesothelial cells in <d>pleural and lung diseases</d>
+<d>Idiopathic pulmonary fibrosis</d>: an altered fibroblast proliferation linked to <d>cancer</d> biology
+Wnt coreceptor <g>Lrp5</g> is a driver of <d>idiopathic pulmonary fibrosis</d>
+The <g>JAK2</g> pathway is activated in <d>idiopathic pulmonary fibrosis</d>
+<g>miR-31</g> is a negative regulator of fibrogenesis and <d>pulmonary fibrosis</d>
+<g>NOX4</g>/NADPH oxidase <e>expression </e>is <v>increased </v>in pulmonary fibroblasts from patients with <d>idiopathic pulmonary fibrosis</d> and mediates <g>TGFbeta1</g>-induced fibroblast differentiation into myofibroblasts
+Matrix regulation of <d>idiopathic pulmonary fibrosis</d>: the role of enzymes
+[The expressions and meanings of <g>BMP-7</g> and <g>TGF-b</g> in <d>idiopathic pulmonary fibrosis</d> and <d>idiopathic nonspecific interstitial pneumonia</d>]
+Mesenchymal Stem Cells Correct Inappropriate Epithelial-mesenchyme Relation in <d>Pulmonary Fibrosis</d>
+Interstitial <d>fibrosis</d> and growth factors
+<u>Blockade </u>of the Wnt/b-catenin pathway attenuates bleomycin-induced <d>pulmonary fibrosis</d>
+Control of virus reactivation arrests pulmonary herpesvirus-induced <d>fibrosis</d> in IFN-gamma receptor-<u>deficient </u>mice
+Anti-pulmonary fibrotic activity of salvianolic acid B was screened by a novel method based on the cyto-biophysical properties
+Cytokines in human <d>lung fibrosis</d>
+<e>Expression </e>of <g>RXFP1</g>
+Association between cytokine removal by polymyxin B hemoperfusion and improved pulmonary oxygenation in patients with acute exacerbation of <d>idiopathic pulmonary fibrosis</d>
+Inhibition of mechanosensitive signaling in myofibroblasts ameliorates experimental <d>pulmonary fibrosis</d>
+y-Herpes virus-68, but not Pseudomonas aeruginosa or influenza A (H1N1), exacerbates <d>established murine lung fibrosis</d>
+Association of HLA and cytokine gene polymorphisms with <d>idiopathic pulmonary fibrosis</d>
+Signaling pathways and their miRNA regulators involved in the etiopathology of <d>idiopathic pulmonary fibrosis</d> (<d>IPF</d>) and <d>hypersensitivity pneumonitis</d> (<d>HP</d>)
+The role of microRNA-155/liver X receptor pathway in experimental and <d>idiopathic pulmonary fibrosis</d>
+<g>Cysteine-rich protein 1</g> is regulated by <g>transforming growth factor-b1</g> and expressed in lung <d>fibrosis</d>

+FAK-related nonkinase is a multifunctional negative regulator of pulmonary fibrosis
+Bleomycin and IL-1beta-mediated pulmonary fibrosis is IL-17A dependent
+Hsp90 regulation of fibroblast activation in pulmonary fibrosis
+Significant involvement of CCL2 (MCP-1) in inflammatory disorders of the lung
+BARD1 mediates TGF-b signaling in pulmonary fibrosis
+Renin is an angiotensin-independent profibrotic mediator: role in pulmonary fibrosis
+Up-regulation of heparan sulfate 6-O-sulfation in idiopathic pulmonary fibrosis
+Mechanisms of fibrosis in coal workers' pneumoconiosis
+Role for alpha3 integrin in EMT and pulmonary fibrosis
+Molecular targets in pulmonary fibrosis: the myofibroblast in focus
+Increased alveolar soluble annexin V promotes lung inflammation and fibrosis
+Re-evaluation of fibrogenic cytokines in lung fibrosis
+A critical role for the mTORC2 pathway in lung fibrosis
+Inhibition and role of let-7d in idiopathic pulmonary fibrosis
+Predisposition for disrepair in the aged lung
+MicroRNA regulatory networks in idiopathic pulmonary fibrosis
+Wnt coreceptor Lrp5 is a driver of idiopathic pulmonary fibrosis
+Pirfenidone inhibits myofibroblast differentiation and lung fibrosis development during insufficient mitophagy
+Transcription factor GATA-6 is expressed in quiescent myofibroblasts in idiopathic pulmonary fibrosis
+[Idiopathic pulmonary fibrosis and lung cancer]
+Role of caveolin-1 in fibrotic diseases
+Caveolin-1: a critical regulator of lung fibrosis in idiopathic pulmonary fibrosis
+Azithromycin attenuates myofibroblast differentiation and lung fibrosis development through proteasomal degradation of NOX4
+[Potential role of cytokines in idiopathic pulmonary fibrosis]
+The potential role of PDGF, IGF-1, TGF-beta expression in idiopathic pulmonary fibrosis
+Cytokine profiles in idiopathic pulmonary fibrosis suggest an important role for TGF-beta and IL-10
+Therapeutic targets in fibrotic pathways
+Profibrotic role of WNT10A via TGF-b signaling in idiopathic pulmonary fibrosis
+Autophagy in idiopathic pulmonary fibrosis
+Overexpression of Sulf2 in idiopathic pulmonary fibrosis
+Matrix metalloproteinase (MMP)-19 deficient fibroblasts display a profibrotic phenotype
+[Pulmonary fibrosis--a therapeutic dilemma?]
+Signaling pathways in the epithelial origins of pulmonary fibrosis
+Participation of miR-200 in pulmonary fibrosis
+Proteasomal regulation of pulmonary fibrosis
+The JAK2 pathway is activated in idiopathic pulmonary fibrosis
+Transforming growth factor-beta1 in sarcoidosis
+Cytokines in human lung fibrosis
+Cartilage oligomeric matrix protein in idiopathic pulmonary fibrosis
+Cytokine gene polymorphisms in idiopathic pulmonary fibrosis
+Matrix metalloproteinase 3 is a mediator of pulmonary fibrosis
+N-acetylcysteine inhibits alveolar epithelial-mesenchymal transition
+Pulmonary fibrosis: pathogenesis, etiology and regulation
+Profibrotic role of miR-154 in pulmonary fibrosis
+IPF lung fibroblasts have a senescent phenotype
+Sphingolipids in pulmonary fibrosis
+Neutrophil elastase promotes myofibroblast differentiation in lung fibrosis
+BALF N-acetylglucosaminidase and beta-galactosidase activities in idiopathic pulmonary fibrosis
+Transglutaminase 2 and its role in pulmonary fibrosis
+The role of cytokines in human lung fibrosis
+Pleural mesothelial cells in pleural and lung diseases
+[Different cytokine profiles in usual interstitial pneumonia and nonspecific interstitial pneumonia]
+Essential role for the ATG4B protease and autophagy in bleomycin-induced pulmonary fibrosis
+Deregulation of selective autophagy during aging and pulmonary fibrosis: the role of TGFb1
+MiR-338* suppresses fibrotic pathogenesis in pulmonary fibrosis through targeting LPA1
+Metformin attenuates lung fibrosis development via NOX4 suppression
+Investigation of bone marrow mesenchymal stem cells (BM MSCs) involvement in Idiopathic Pulmonary Fibrosis (IPF)
+The pathogenesis of idiopathic pulmonary fibrosis
+Interstitial fibrosis and growth factors
+TIAM1 inhibits lung fibroblast differentiation in pulmonary fibrosis
+Role of von Hippel-Lindau protein in fibroblast proliferation and fibrosis
+miR-21 mediates fibrogenic activation of pulmonary fibroblasts and lung fibrosis
+miR-31 is a negative regulator of fibrogenesis and pulmonary fibrosis
+TRPV4 mediates myofibroblast differentiation and pulmonary fibrosis in mice
+Matrix regulation of idiopathic pulmonary fibrosis: the role of enzymes
+Epithelial-mesenchymal transition in chronic hypersensitivity pneumonitis
+Inhibition of mechanosensitive signaling in myofibroblasts ameliorates experimental pulmonary fibrosis
+[A role for mesothelial cells in the genesis of idiopathic pulmonary fibrosis?]
+Pathogenesis of idiopathic pulmonary fibrosis
+TGF-b activation and lung fibrosis
+Protective role of andrographolide in bleomycin-induced pulmonary fibrosis in mice
+[The potential role of cytokines expression in idiopathic pulmonary fibrosis]
+Autophagy and inflammation in chronic respiratory disease
+Reviews and prospectives of signaling pathway analysis in idiopathic pulmonary fibrosis
+Membrane type-matrix metalloproteinases in idiopathic pulmonary fibrosis
+EMT and interstitial lung disease: a mysterious relationship
+Fibrosis of two: Epithelial cell-fibroblast interactions in pulmonary fibrosis
+Lysyl oxidases regulate fibrillar collagen remodelling in idiopathic pulmonary fibrosis
+MicroRNAs in idiopathic pulmonary fibrosis
+Yin yang 1 is a novel regulator of pulmonary fibrosis

+M2 macrophages <v>induce </v><g>EMT</g> through the <g>TGF-b</g>/<g>Smad2</g> signaling pathway
+Up-regulation of heparan sulfate 6-O-sulfation in <d>idiopathic pulmonary fibrosis</d>
+Pirfenidone attenuates bleomycin-induced pulmonary fibrosis in mice by <r>regulating </r><g>Nrf2</g>/<g>Bach1</g> equilibrium
+Signalling pathways from NADPH oxidase-4 to <d>idiopathic pulmonary fibrosis</d>
+The Role of PPARs in Lung <d>Fibrosis</d>
+Free radical generation induces epithelial-to-mesenchymal transition in lung epithelium via a <g>TGF-b1</g>-dependent mechanism
+<v>Overexpression </v>of <g>Sulf2</g> in <d>idiopathic pulmonary fibrosis</d>
+Essential role for the <g>ATG4B</g> protease and autophagy in bleomycin-induced <d>pulmonary fibrosis</d>
+Smooth muscle alpha-actin <e>expression </e>and myofibroblast differentiation by <g>TGFbeta</g> are <v>dependent </v>upon <g>MK2</g>
+<v>Elevated </v><g>sL1</g>-CAM levels in BALF and serum of <d>IPF</d> patients
+Microarray identifies ADAM family members as key responders to <g>TGF-beta1</g> in alveolar epithelial cells
+Titration of non-replicating adenovirus as a vector for <v>transducing </v>active <g>TGF-beta1</g> gene <e>expression </e>causing <d>inflammation</d> and fibrogenesis in the lungs of C57BL/6 mice
+A critical role for the <g>mTORC2</g> pathway in <d>lung fibrosis</d>
+<g>TGF-beta</g> driven lung <d>fibrosis</d> is macrophage dependent and blocked by Serum amyloid P
+Sputum biomarkers in <d>IPF</d>: Evidence for raised gene <e>expression </e>and protein level of <g>IGFBP-2</g>, <g>IL-8</g> and <g>MMP-7</g>
+<g>Forkhead Box F1</g> (<g>FOXF1</g>) <u>represses </u>cell growth, COL1 and <g>ARPC2</g> <e>expression </e>in lung fibroblasts in vitro
+<g>MicroRNA-29c</g> regulates apoptosis sensitivity via modulation of the cell-surface death receptor, Fas, in lung fibroblasts
+<g>Prostaglandin F(2alpha) receptor</g> signaling facilitates bleomycin-induced <d>pulmonary fibrosis</d> independently of transforming growth factor-beta
+Cytoskeletal protein modulation in <d>pulmonary alveolar myofibroblasts</d> during <d>idiopathic pulmonary fibrosis</d>
+<g>Transglutaminase 2</g> and its role in <d>pulmonary fibrosis</d>
+Sunitinib, a Small-Molecule Kinase <u>Inhibitor,</u>
+<g>WISP1</g> mediates <g>IL-6</g>-dependent proliferation in primary human lung fibroblasts
+<v>Upregulation </v>of alveolar <e>levels </e>of activin B, but not activin A, in lungs of west highland white terriers with <d>idiopathic pulmonary fibrosis</d> and <d>diffuse alveolar damage</d>
+Absence of <g>Thy-1</g> results in <g>TGF-b</g> induced <g>MMP-9</g> expression and confers a profibrotic phenotype to human lung fibroblasts
+Shikonin suppresses pulmonary fibroblasts proliferation and activation by <r>regulating </r><g>Akt</g> and <g>p38</g>
+<g>TRPV4</g> mediates myofibroblast differentiation and <d>pulmonary fibrosis</d> in mice
+Modulation of <g>CD11c</g>+ lung dendritic cells in respect to <g>TGF-b</g> in experimental <d>pulmonary fibrosis</d>
+Induction of epithelial-mesenchymal transition in alveolar epithelial cells by <g>transforming growth factor-beta1</g>: potential role in <d>idiopathic pulmonary fibrosis</d>
+Cytokine gene polymorphisms in <d>idiopathic pulmonary fibrosis</d>
+<g>Cub domain containing protein 1</g> (<g>CDCP1</g>) negatively regulates TGFb signaling and myofibroblast differentiation
+Increased levels of prostaglandin E-major urinary metabolite (PGE-MUM) in chronic fibrosing <d>interstitial pneumonia</d>
+Mechanisms of <d>fibrosis</d> in <d>coal workers' pneumoconiosis</d>
+Different effects of growth factors on proliferation and matrix production of normal and fibrotic human lung fibroblasts
+<g>miR</g>-323a-3p regulates <d>lung fibrosis</d> by targeting multiple profibrotic pathways
+<g>TGF-b1</g> <v>stimulates </v><g>HDAC4</g> nucleus-to-cytoplasm <l>translocation </l>and <g>NADPH oxidase 4</g>-derived reactive oxygen species in normal human lung fibroblasts
+MicroRNA-326 regulates profibrotic functions of transforming growth factor-b in <d>pulmonary fibrosis</d>
+Pathogenesis of <d>idiopathic pulmonary fibrosis</d>
+Nitric oxide attenuates epithelial-mesenchymal transition in alveolar epithelial cells
+<e>Expression </e>of <g>WNT5A</g> in <d>Idiopathic Pulmonary Fibrosis</d> and Its <r>Control </r>by <g>TGF-b</g> and <g>WNT7B</g> in Human Lung Fibroblasts
+<g>FGF-1</g> reverts epithelial-mesenchymal transition induced by <g>TGF-{beta}1</g> through MAPK/ERK kinase pathway
+Type II alveolar epithelial cells and interstitial fibroblasts express <g>connective tissue growth factor</g> in <d>IPF</d>
+<g>Caveolin-1</g>: a critical regulator of <d>lung fibrosis</d> in <d>idiopathic pulmonary fibrosis</d>
+<g>Cthrc1</g> lowers pulmonary collagen associated with bleomycin-induced <d>fibrosis</d> and protects lung function
+Protective Effect of Ginsenoside Rg1 on Bleomycin-Induced Pulmonary Fibrosis in Rats: Involvement of <g>Caveolin-1</g> and <g>TGF-b1</g>
+<u>Defect </u>of <g>hepatocyte growth factor</g> <l>secretion </l>by fibroblasts in <d>idiopathic pulmonary fibrosis</d>
+Rapamycin <v>increases </v><g>CCN2</g> <e>expression </e>of lung fibroblasts via <g>phosphoinositide 3-kinase</g>
+Human lung myofibroblast <g>TGFb1</g>-dependent <g>Smad2/3</g> signalling is Ca(2+)-dependent and <r>regulated </r>by <g>KCa3.1</g>
+[A role for mesothelial cells in the genesis of <d>idiopathic pulmonary fibrosis</d>?]
+Antifibrotic effects of <g>cyclosporine A</g> on <g>TGF-b1</g>-treated lung fibroblasts and lungs from bleomycin-treated mice: role of <g>hypoxia-inducible factor-1a</g>
+<g>TGF-beta 1</g> as an enhancer of Fas-mediated apoptosis of lung epithelial cells
+Proliferation of pulmonary interstitial fibroblasts is mediated by <g>transforming growth factor-beta1</g>-induced release of extracellular <g>fibroblast growth factor-2</g> and <p>phosphorylation </p>of <g>p38</g>
+Prognostic factors for <d>idiopathic pulmonary fibrosis</d>: clinical, physiologic, pathologic, and molecular aspects
+Hydrogen peroxide is a diffusible paracrine signal for the induction of epithelial cell death by activated myofibroblasts
+<g>Secreted protein acidic and rich in cysteine</g> (<g>SPARC</g>) is upregulated by transforming growth factor (TGF)-b and is required for <g>TGF-b</g>-induced hydrogen peroxide production in fibroblasts
+The lncRNA <g>H19</g>
+<e>Contribution </e>of the anaphylatoxin receptors, <g>C3aR</g> and <g>C5aR</g>, to the pathogenesis of <d>pulmonary fibrosis</d>
+Corilagin attenuates aerosol bleomycin-induced experimental <d>lung injury</d>
+[Potential role of cytokines in <d>idiopathic pulmonary fibrosis</d>]
+Protective role of andrographolide in bleomycin-induced <d>pulmonary fibrosis</d> in mice
+<g>Serpin B4</g> isoform overexpression is associated with aberrant epithelial proliferation and <d>lung cancer</d> in <d>idiopathic pulmonary fibrosis</d>
+Interplay between <g>RAGE</g>, <g>CD44</g>, and focal adhesion molecules in epithelial-mesenchymal transition of alveolar epithelial cells
+Antifibrotic properties of receptor for advanced glycation end products in <d>idiopathic pulmonary fibrosis</d>
+Dysregulated Collagen Homeostasis by Matrix Stiffening and <g>TGF-b1</g> in Fibroblasts from <d>Idiopathic Pulmonary Fibrosis</d>
+TGF-b1 T869C polymorphism may affect susceptibility to <d>idiopathic pulmonary fibrosis</d> and disease severity
+Plasma <g>CCN2</g> (connective tissue growth factor; <g>CTGF</g>) is a potential biomarker in <d>idiopathic pulmonary fibrosis</d> (<d>IPF</d>)
+Azithromycin attenuates myofibroblast differentiation and lung <d>fibrosis</d> development through <d>proteasomal degradation</d> of <g>NOX4</g>
+Crosstalk between <g>TGF-b1</g> and complement activation augments epithelial <d>injury in pulmonary fibrosis</d>
+Bleomycin and <g>IL-1beta</g>-mediated <d>pulmonary fibrosis</d> is <g>IL-17A</g> dependent
+miR-199a-5p Is <v>upregulated </v>during fibrogenic response to tissue injury and mediates TGFbeta-induced lung fibroblast activation by targeting <g>caveolin-1</g>
+Integrated analyses identify the involvement of microRNA-26a in epithelial-mesenchymal transition during <d>idiopathic pulmonary fibrosis</d>
+<g>Transforming growth factor beta1</g> induces epithelial-to-mesenchymal transition of A549 cells
+The anti-fibrotic effects of microRNA-153 by <r>targeting </r><g>TGFBR-2</g> in <d>pulmonary fibrosis</d>
+<u>Defective </u>histone acetylation is <r>responsible </r>for the <u>diminished </u><e>expression </e>of <g>cyclooxygenase 2</g> in <d>idiopathic pulmonary fibrosis</d>
+Inhibition and role of <g>let-7d</g> in <d>idiopathic pulmonary fibrosis</d>
+Pleural mesothelial cell transformation into myofibroblasts and haptotactic migration in response to <g>TGF-beta1</g> in vitro
+<g>miR-21</g> mediates fibrogenic activation of pulmonary fibroblasts and lung <d>fibrosis</d>
+Inhibitory effects of amines from Citrus reticulata on bleomycin-induced <d>pulmonary fibrosis</d> in rats
+Thalidomide <u>reduces </u><g>IL-18</g>, <g>IL-8</g> and <g>TNF-alpha</g> <l>release </l>from alveolar macrophages in <d>interstitial lung disease</d>
+Effect of glycosides based standardized fenugreek seed extract in bleomycin-induced <d>pulmonary fibrosis</d> in rats: Decisive role of <g>Bax</g>, <g>Nrf2</g>, <g>NF-kB</g>, <g>Muc5ac</g>, <g>TNF-a</g> and <g>IL-1b</g>
+The Anti-fibrotic Effects and Mechanisms of MicroRNA-486-5p in <d>Pulmonary Fibrosis</d>

@@ -7,4 +7,16 @@ Partition	Cluster	GeneDisease	Total
 4	1	17	48
 4	1	17	48
 4	2	90	229
 4	2	90	229
 4	3	25	42
 4	3	25	42
-4	4	46	132
\ No newline at end of file
+4	4	46	132
+5	1	17	48
+5	2	49	104
+5	3	41	125
+5	4	25	42
+5	5	46	132
+6	1	17	48	
+6	2	49	104
+6	3	41	125	
+6	4	25	42
+6	5	15	52
+6	6	31	80
+