So , it is possible that OmpR independently regulate the expression of tviA in S. typhi at osmotic-stress .
Acquisition of the tviA gene by S. Typhi integrated the regulation of flagella into the OmpR regulons , such that flagellum expression is decreased following invasion of the intestinal mucosa , thereby avoiding detection by TLR5 present on the basolateral surface of CD11c dendritic cells in the lamina .
Acquisition of the tviA gene by S. Typhi integrated the regulation of flagella into the OmpR regulons , such that flagellum expression is decreased following invasion of the intestinal mucosa , thereby avoiding detection by TLR5 present on the basolateral surface of enterocytes dendritic cells in the lamina .
OmpR binds to the promoters of tviA to activate their transcription ; however , it negatively regulates hfq in an indirect manner .
OmpR binds to the promoters of tviA to activate their transcription ; however , it positively regulates tviB .
: -LRB- 0123456789 OmpR also binds to the promoter of tviA to activate its transcription , and thus act as an activator of Vi antigen .
These results indicated that the transcription of tviA was under negative control of OmpR , respectively .
These results indicated that the transcription of tviA was under positive control of OmpR , respectively .
Thus , OmpR only directly regulates two , i.e. , tviA , of the four genes .