The role of the alternative cf factor RpoS in the regulation of the starvation survival loci , stiB , has been characterized .
In contrast , RpoS was found to be required for the negative regulation of stiB during logarithmic phase .
In contrast , RpoS was found to be required for the negative regulation of stiB during C starvation-induced stationary-phase .
In contrast , RpoS was found to be required for the negative regulation of stiB during P starvation-induced stationary-phase .
RpoS functions in the negative regulation of stiB .
The rpoS mutation resulted in a two-to threefoldhigher level of induction of a fiveto sixfold-higher level of induction during C starvation , suggesting that RpoS is involved in the negative regulation of the stiB locus .
The rpoS mutation resulted in a two-to threefoldhigher level of induction of stiB during P starvation during C starvation , suggesting that RpoS is involved in the negative regulation of the stiB locus .
The fact that RpoS is an alternative a factor suggests that rpoS controls the expression of a negative regulator of stiB .
Since stiB are regulated by RpoS , we wanted to examine the combined effects of sti mutations on starvation survival .
Since stiB are regulated by RpoS , we wanted to examine the combined effects of rpoS mutations on starvation survival .
Role of the alternative a factor RpoS , or a 
On the basis of the fact that stiB are all regulated by RpoS , one might predict that rpoS mutants exhibit increased sensitivity to the effects of C starvation because stiC are not induced and/or because stiB is overexpressed in an rpoS mutant .
On the basis of the fact that stiB are all regulated by RpoS , one might predict that rpoS mutants exhibit increased sensitivity to the effects of C starvation because stiA are not induced and/or because stiB is overexpressed in an rpoS mutant .