Finally , the identification of a SlyA binding site suggests a mechanism to explain how SlyA overproduction enhances hlyE expression by antagonizing the negative effects of H-NS .
In contrast , in the absence of hns , SlyA overproduction did not enhance hlyE expression .
indirectly , SlyA activates hlyE expression ultimately by counteracting the negative effects of H-NS
indirectly , SlyA activates hlyE expression by modulating H-NS binding
directly , SlyA activates hlyE expression ultimately by counteracting the negative effects of H-NS
directly , SlyA activates hlyE expression by modulating H-NS binding
in-vitro evidence suggests that SlyA activates hlyE expression by antagonizing H-NS-mediated repression .
In vivo suggests that SlyA activates hlyE expression by antagonizing H-NS-mediated repression .
that SlyA does not enhance hlyE expression in hns cultures
SlyA activates hlyE expression by antagonising H-NS-mediated repression .
It was reported that hlyE could be activated by SlyA , an important regulator of virulence genes in Salmonella .