These results showed that , whereas OmpR-mediated regulation of hilA expression was dampened in a DhilC DrtsA background , regulation was blocked only in strains missing HilD , suggesting that OmpR controls SPI1 expression by regulating hilD expression .
Increased OmpR can be recruited to bind to hilD promoters , in a process .
The OmpR protein also controls the transcription of key regulatory genes in hilD , allowing OmpR to contribute to the coordination of expression of these major virulence genetic elements .