These findings suggest that STM1697 inhibits motility by interacting at the post-transcriptional level with the FlhD C functionality as demonstrated previously for STM1344 .
These findings suggest that STM1697 inhibits motility by interacting at the post-transcriptional level with the FlhD C 4 2 expression .
The expression of the FlhD-free mutants of STM1697 showed similar motility phenotypes as the stm1697 strain suggesting that the motility inhibition of STM1697 is directly mediated by interaction with FlhD .