In accord with these data , another group showed that dalS/STM1633 clustered into an SsrB-controlled regulatory network enriched in virulence genes including genes in SPI-2 and with srfN ( 19 ) , the latter of which we previously showed was required for in-vivo fitness ( 20 ) .
SsrB controls srfN directly through binding to the srfN cis-regulatory element
SsrB controls srfN directly through binding to the srfN cis-regulatory element
SsrB controls srfN directly through binding to the srfN cis-regulatory element