Our model suggests that SirA could control expression of hilD by regulating the expression of a single regulator .
Our model suggests that SirA could control expression of hilD by either independently activating each gene .
SirA apparently controls hilD at the level of the mRNA ; overproduction of SirA increased hilD expression in a HilD-independent fashion .
This genetic analysis is apparently inconsistent with previous gel shift data suggesting that SirA binds the hilD .
Conversely , previously published gel-shift data suggested that SirA is able to bind to that of hilD .