In contrast , absence of H-NS showed 5.3-fold increase of hilA expression under low-osmolarity conditions .
in antagonizing H-NS silencing of hilA to promote its Stationary-phase induction of hilA in strain expression under permissive conditions .
Whereas hilA induction at antagonize H-NS hilA silencing account for hilA derepres-the onset of the stationary-phase should be abolished in sion in cells growing in LB-medium at 37 uC .
Whereas hilA induction at antagonize H-NS hilA silencing account for hilA derepres-the onset of the stationary-phase should be abolished in sion in cells entering mutants .
Interestingly , a partial inhibition of H-NS activity in cells resulted in a signi-ficant induction of hilA expression , up to about 50 % of the expression .