the SOS response .4,5 The initial response to DNA damage is induction of LexA repressed genes include dinP
dinP are two names for one gene , there is no dinB in S. typhimurium .8 ) The sbmC gene was originally identified by its ability to confer resistance to MccB17 at high copy number .9 MccB17 is a peptide antibiotic , possesses much like umuDC , the expression of sbmC is LexA dependent .9 Thus , upregulation of dinP by all nine FQs is likely LexA dependent .
dinP are two names for one gene , there is no dinB in S. typhimurium .8 ) The sbmC gene was originally identified by its ability to confer resistance to MccB17 at high copy number .9 MccB17 is a peptide antibiotic , possesses much like umuDC , the expression of sbmC is RecA dependent .9 Thus , upregulation of dinP by all nine FQs is likely LexA dependent .
dinP are two names for one gene , there is no dinB in S. typhimurium .8 ) The sbmC gene was originally identified by its ability to confer resistance to MccB17 at high copy number .9 MccB17 is a peptide antibiotic , possesses a quasicanonical LexA box , the expression of sbmC is LexA dependent .9 Thus , upregulation of dinP by all nine FQs is likely LexA dependent .
dinP are two names for one gene , there is no dinB in S. typhimurium .8 ) The sbmC gene was originally identified by its ability to confer resistance to MccB17 at high copy number .9 MccB17 is a peptide antibiotic , possesses a quasicanonical LexA box , the expression of sbmC is RecA dependent .9 Thus , upregulation of dinP by all nine FQs is likely LexA dependent .
dinB are two names for one gene , there is no dinB in S. typhimurium .8 ) The sbmC gene was originally identified by its ability to confer resistance to MccB17 at high copy number .9 MccB17 is a peptide antibiotic , possesses much like umuDC , the expression of sbmC is LexA dependent .9 Thus , upregulation of dinP by all nine FQs is likely LexA dependent .
dinB are two names for one gene , there is no dinB in S. typhimurium .8 ) The sbmC gene was originally identified by its ability to confer resistance to MccB17 at high copy number .9 MccB17 is a peptide antibiotic , possesses much like umuDC , the expression of sbmC is RecA dependent .9 Thus , upregulation of dinP by all nine FQs is likely LexA dependent .
dinB are two names for one gene , there is no dinB in S. typhimurium .8 ) The sbmC gene was originally identified by its ability to confer resistance to MccB17 at high copy number .9 MccB17 is a peptide antibiotic , possesses a quasicanonical LexA box , the expression of sbmC is LexA dependent .9 Thus , upregulation of dinP by all nine FQs is likely LexA dependent .
dinB are two names for one gene , there is no dinB in S. typhimurium .8 ) The sbmC gene was originally identified by its ability to confer resistance to MccB17 at high copy number .9 MccB17 is a peptide antibiotic , possesses a quasicanonical LexA box , the expression of sbmC is RecA dependent .9 Thus , upregulation of dinP by all nine FQs is likely LexA dependent .
coli are two names for one gene , there is no dinB in S. typhimurium .8 ) The sbmC gene was originally identified by its ability to confer resistance to MccB17 at high copy number .9 MccB17 is a peptide antibiotic , possesses much like umuDC , the expression of sbmC is LexA dependent .9 Thus , upregulation of dinP by all nine FQs is likely LexA dependent .
coli are two names for one gene , there is no dinB in S. typhimurium .8 ) The sbmC gene was originally identified by its ability to confer resistance to MccB17 at high copy number .9 MccB17 is a peptide antibiotic , possesses much like umuDC , the expression of sbmC is RecA dependent .9 Thus , upregulation of dinP by all nine FQs is likely LexA dependent .
coli are two names for one gene , there is no dinB in S. typhimurium .8 ) The sbmC gene was originally identified by its ability to confer resistance to MccB17 at high copy number .9 MccB17 is a peptide antibiotic , possesses a quasicanonical LexA box , the expression of sbmC is LexA dependent .9 Thus , upregulation of dinP by all nine FQs is likely LexA dependent .
coli are two names for one gene , there is no dinB in S. typhimurium .8 ) The sbmC gene was originally identified by its ability to confer resistance to MccB17 at high copy number .9 MccB17 is a peptide antibiotic , possesses a quasicanonical LexA box , the expression of sbmC is RecA dependent .9 Thus , upregulation of dinP by all nine FQs is likely LexA dependent .